Mechanism of Siglec-8-induced human eosinophil apoptosis: Role of caspases and mitochondrial injury
- 28 October 2005
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 336 (3) , 918-924
- https://doi.org/10.1016/j.bbrc.2005.08.202
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Ligation of Siglec-8: a selective mechanism for induction of human eosinophil apoptosisBlood, 2003
- Molecular Characterization of a Siglec8 Variant Containing Cytoplasmic Tyrosine-Based Motifs, and Mapping of the Siglec8 GeneBiochemical and Biophysical Research Communications, 2000
- Siglec-8Journal of Biological Chemistry, 2000
- The Protein Import Motor of MitochondriaCell, 1999
- Ordering the Cytochrome c–initiated Caspase Cascade: Hierarchical Activation of Caspases-2, -3, -6, -7, -8, and -10 in a Caspase-9–dependent MannerThe Journal of cell biology, 1999
- Diphenyleneiodonium, an NAD(P)H Oxidase Inhibitor, also Potently Inhibits Mitochondrial Reactive Oxygen Species ProductionBiochemical and Biophysical Research Communications, 1998
- The major SHP-1-binding, tyrosine-phosphorylated protein in macrophages is a member of the KIR/LIR family and an SHP-1 substrateOncogene, 1998
- The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell deathOncogene, 1997
- Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo.The Journal of Experimental Medicine, 1995
- Mechanism of O−2 generation in reduction and oxidation cycle of ubiquinones in a model of mitochondrial electron transport systemsBiochimica et Biophysica Acta (BBA) - Bioenergetics, 1988