Pharmacological complement inhibition at the C3 convertase level promotes neuronal survival, neuroprotective intracerebral gene expression, and neurological outcome after traumatic brain injury
- 1 June 2006
- journal article
- research article
- Published by Elsevier in Experimental Neurology
- Vol. 199 (2) , 454-464
- https://doi.org/10.1016/j.expneurol.2006.01.033
Abstract
No abstract availableKeywords
This publication has 56 references indexed in Scilit:
- Effect of atorvastatin on spatial memory, neuronal survival, and vascular density in female rats after traumatic brain injuryJournal of Neurosurgery, 2005
- Erythropoietin is neuroprotective, improves functional recovery, and reduces neuronal apoptosis and inflammation in a rodent model of experimental closed head injuryThe FASEB Journal, 2005
- Closed head injury—an inflammatory disease?Brain Research Reviews, 2005
- Selective Induction of Apoptosis in Melanoma Cells by Tyrosinase Promoter-Controlled CD95 Ligand OverexpressionJournal of Investigative Dermatology, 2005
- Neuroprotection from Complement‐Mediated Inflammatory DamageAnnals of the New York Academy of Sciences, 2004
- Activation of Complement in the Central Nervous SystemAnnals of the New York Academy of Sciences, 2003
- Vaccinia Virus Complement Control Protein Enhances Functional Recovery after Traumatic Brain InjuryJournal of Neurotrauma, 2002
- The Phenomenology of Personality Change Due to Traumatic Brain Injury in Children and AdolescentsThe Journal of Neuropsychiatry and Clinical Neurosciences, 2001
- Closed Head Injury Triggers Early Production of TNFα and IL-6 by Brain TissueJournal of Cerebral Blood Flow & Metabolism, 1994
- Mouse Crry/p65 is a regulator of the alternative pathway of complement activationEuropean Journal of Immunology, 1993