Mitochondrial KATPChannel as an End Effector of Cardioprotection During Late Preconditioning: Triggering Role of Nitric Oxide
- 1 November 2001
- journal article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 33 (11) , 2037-2046
- https://doi.org/10.1006/jmcc.2001.1468
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Nitric Oxide Inhibition Improved Myocardial Metabolism Independent of Tissue Perfusion During Ischemia But Not During ReperfusionJournal of Molecular and Cellular Cardiology, 2000
- Infarct Size and Nitric Oxide Synthase in Murine MyocardiumJournal of Molecular and Cellular Cardiology, 2000
- Characterization of protein kinase C beta isoform activation on the gene expression of transforming growth factor-beta, extracellular matrix components, and prostanoids in the glomeruli of diabetic rats.Journal of Clinical Investigation, 1997
- THE NF-κB AND IκB PROTEINS: New Discoveries and InsightsAnnual Review of Immunology, 1996
- Nitric oxide signaling in ischemic heartCardiovascular Research, 1995
- NF-κB: A lesson in family valuesCell, 1995
- Sustain inhibition of nitric oxide by NG-nitro-l-arginine improves myocardial function following ischemia/reperfusion in isolated perfused rat heartJournal of Molecular and Cellular Cardiology, 1995
- Role of ATP sensitive potassium channels in long term adaptation to metabolic stressCardiovascular Research, 1994
- Promoter of the mouse gene encoding calcium-independent nitric oxide synthase confers inducibility by interferon gamma and bacterial lipopolysaccharide.The Journal of Experimental Medicine, 1993
- Increased endothelial albumin permeability mediated by protein kinase C activation.Journal of Clinical Investigation, 1990