Oral administration of homocysteine leads to increased plasma triglycerides and homocysteic acid — additional mechanisms in homocysteine induced endothelial damage?
- 14 July 1995
- journal article
- Published by Elsevier in Life Sciences
- Vol. 57 (8) , 813-817
- https://doi.org/10.1016/0024-3205(95)02009-8
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Carotid artery intimal-medial wall thickening and plasma homocyst(e)ine in asymptomatic adults. The Atherosclerosis Risk in Communities Study.Circulation, 1993
- Adverse vascular effects of homocysteine are modulated by endothelium-derived relaxing factor and related oxides of nitrogen.Journal of Clinical Investigation, 1993
- Hyperhomocysteinaemia in stroke: prevalence, cause, and relationships to type of stroke and stroke risk factorsEuropean Journal of Clinical Investigation, 1992
- High-performance liquid chromatographic method with fluorescence detection for the determination of total homocyst(e)ine in plasmaJournal of Chromatography A, 1992
- Arterial wall oxygenation, oxyradicals, and atherosclerosisAtherosclerosis, 1991
- Hyperhomocysteinemia: An Independent Risk Factor for Vascular DiseaseNew England Journal of Medicine, 1991
- Elevated plasma homocyst(e)ine concentration as a possible independent risk factor for stroke.Stroke, 1990
- Prevalence of hyperhomocyst(e)inemia in patients with peripheral arterial occlusive disease.Circulation, 1989
- Endothelial cell injury due to copper-catalyzed hydrogen peroxide generation from homocysteine.Journal of Clinical Investigation, 1986
- Moderate homocysteinemia--a possible risk factor for arteriosclerotic cerebrovascular disease.Stroke, 1984