Lesion of dopaminergic terminals in the amygdala produces enhanced locomotor response to d-amphetamine and opposite changes in dopaminergic activity in prefrontal cortex and nucleus accumbens
- 1 May 1988
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 447 (2) , 335-340
- https://doi.org/10.1016/0006-8993(88)91136-5
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Increased sensitivity to amphetamine and facilitation of amphetamine self-administration after 6-hydroxydopamine lesions of the amygdalaPsychopharmacology, 1988
- Laterality of dopamine function and neuroleptic action in the amygdala in the ratNeuropharmacology, 1985
- Dopaminergic supersensitivity follows ferric chloride-induced limbic seizuresBiological Psychiatry, 1985
- Locomotor behavior following kindling in three different brain sitesBrain Research, 1985
- Post-ictal locomotor activity in three different rat models of epilepsyBrain Research, 1982
- Response to stress of mesocortico-frontal dopaminergic neurones in rats after long-term isolationNature, 1980
- Modification of dopaminergic receptor sensitivity in rat brain after amygdaloid kindlingExperimental Neurology, 1979
- Long-lasting depletion of dopamine in the rat amygdala induced by kindling stimulationBrain Research, 1977
- ISOLATION AND RADIOENZYMIC ESTIMATION OF PICOGRAM QUANTITIES OF DOPAMINE AND NOREPINEPHRINE IN BIOLOGICAL SAMPLESJournal of Neurochemistry, 1976
- The behavioural effects of dopamine applied intracerebrally to areas of the mesolimbic systemEuropean Journal of Pharmacology, 1975