Demonstrating Alcoholic Cirrhosis of the Liver by Tc-99m BIDA Scintigram

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Abstract
Six patients with decompensated cirrhosis of the liver underwent 99mTc BIDA [butylacetanilidoiminodiacetic acid] studies. All demonstrated persistently high blood pool activity in the heart, lung and soft tissue, slow hepatic tracer uptake, prolonged liver-to-bowel transit time and visualization of an enlarged spleen. Four of the 6 patients demonstrated evidence of ascites and in 1 patient there were visible collateral veins of the abdomen. These findings are due primarily to hepatic dysfunction and retaining Tc-99m BIDA in blood pool because of Tc-99m BIDA exclusively hepatic excretion and little or no alternative renal excretion. All 6 99mTc sulfur colloid studies were performed concomitantly. Except for bone marrow uptake and reversal of the normal liver-spleen ratio of radioactivity, the imaging abnormalities observed with 99mTc BIDA were similar to those seen by Tc-99m SC it is concluded that with 99mTc BIDA studies, 3 of 6 abnormal findings, as described, suggest a decompensated stage of cirrhosis of the liver.

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