Peripheral Block of the Hyperpolarization-Activated Cation Current (Ih) Reduces Mechanical Allodynia in Animal Models of Postoperative and Neuropathic Pain
- 1 May 2005
- journal article
- Published by BMJ in Regional Anesthesia & Pain Medicine
- Vol. 30 (3) , 243-248
- https://doi.org/10.1097/00115550-200505000-00006
Abstract
No abstract availableThis publication has 22 references indexed in Scilit:
- Expression of hyperpolarization‐activated and cyclic nucleotide‐gated cation channel subunit 2 in axon terminals of peptidergic nociceptive primary sensory neurons in the superficial spinal dorsal horn of ratsEuropean Journal of Neuroscience, 2004
- Nerve excitability properties in Charcot-Marie-Tooth disease type 1ABrain, 2004
- Assessing the role of Ih channels in synaptic transmission and mossy fiber LTPProceedings of the National Academy of Sciences, 2002
- A specific blocker reveals the presence and function of the hyperpolarization-activated cation current Ih in peripheral mammalian nerve fibresNeuroscience, 1997
- Characterization of a rat model of incisional painPAIN®, 1996
- Neurologic Evaluation of the Rat during Sciatic Nerve Block with LidocaineAnesthesiology, 1995
- Bradykinin-induced ion currents in cultured rat trigeminal ganglion cellsNeuroscience Research, 1993
- Properties of a hyperpolarization‐activated cation current and its role in rhythmic oscillation in thalamic relay neurones.The Journal of Physiology, 1990
- A novel behavioral model of neuropathic pain disorders produced in rats by partial sciatic nerve injuryPAIN®, 1990
- Current‐clamp analysis of a time‐dependent rectification in rat optic nerve.The Journal of Physiology, 1990