Plasmalemmal Modifications of Entamoeba histolytica In vivo

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Abstract
EM was used to study E. histolytica trophozoites in [human] rectal liver lesions. The amoebal surface had bleblike structures and subplasmalemmal vacuoles and plasmalemmal extensions generally similar to the surface lysosome and trigger previously described. Serial sections showed that plasmalemmal extensions were dendritic complexes enfolding the membrane of the subplasmalemmal vacuole and extending to contact host tissue. Membrane-bound cytotoxic hydrolases probably enter the plasmalemmal extension via the translocation of the subplasmalemmal vacuole membrane and are available to act at contact sites. The small blebs may provide an alternate method for membrane-bound hydrolase activity.

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