Metabolism of phenytoin and covalent binding of reactive intermediates in activated human neutrophils

This publication has 47 references indexed in Scilit:

• Bioactivation of xenobiotics by prostaglandin H synthase
  Chemico-Biological Interactions, 1991
• Effects of the tumor promoter 12-O-tetradecanoylphorbol-13-acetate on phenytoin-induced embryopathy in mice
  Toxicology and Applied Pharmacology, 1989
• Tissue Destruction by Neutrophils
  New England Journal of Medicine, 1989
• Modulation of phenytoin teratogenicity and embryonic covalent binding by acetylsalicylic acid, caffeic acid, and α-phenyl-N-t-butylnitrone: Implications for bioactivation by prostaglandin synthetase
  Toxicology and Applied Pharmacology, 1989
• The iron-binding and hydroxyl radical scavenging action of anti-inflammatory drugs
  Xenobiotica, 1988
• Neutrophils in Human Diseases
  New England Journal of Medicine, 1987
• Superoxide‐dependent production of hydroxyl radical catalyzed by iron—EDTA complex
  FEBS Letters, 1978
• Reaction of hydroxyl radicals with benzoic acid. Isomer distribution in the radical intermediates
  The Journal of Physical Chemistry, 1975
• Intoxication with 5,5-diphenylhydantoin (Dilantin): Clinical features, blood levels, urinary metabolites, and metabolic changes in a child
  The Journal of Pediatrics, 1972
• A new metabolite of 5,5-diphenylhydantoin (Dilantin++Dilantin is the Parke-Davis trade name for 5,5-diphenylhydatoin (phenytoin).)
  Biochemical and Biophysical Research Communications, 1970