T-Cell Properties Determine Disease Site, Clinical Presentation, and Cellular Pathology of Experimental Autoimmune Encephalomyelitis
- 30 November 2004
- journal article
- Published by Elsevier
- Vol. 165 (5) , 1519-1533
- https://doi.org/10.1016/s0002-9440(10)63410-4
Abstract
No abstract availableKeywords
This publication has 64 references indexed in Scilit:
- Interferon γ Is Required for Activation-induced Death of T LymphocytesThe Journal of Experimental Medicine, 2002
- The neutrophil as a cellular source of chemokinesImmunological Reviews, 2000
- Failure to Suppress the Expansion of the Activated Cd4 T Cell Population in Interferon γ–Deficient Mice Leads to Exacerbation of Experimental Autoimmune EncephalomyelitisThe Journal of Experimental Medicine, 2000
- TNF-α and IFN-γ render microglia sensitive to Fas ligand-induced apoptosis by induction of Fas expression and down-regulation of Bcl-2 and Bcl-xLEuropean Journal of Immunology, 1998
- Exon 2 containing myelin basic protein (MBP) transcripts are expressed in lesions of experimental allergic encephalomyelitis (EAE)Journal of Neuroimmunology, 1997
- Interferon‐γ confers resistance to experimental allergic encephalomyelitisEuropean Journal of Immunology, 1996
- Antigen-specific down-regulation of myelin basic protein-reactive T cells during spontaneous recovery from experimental autoimmune encephalomyelitis: further evidence of apoptotic deletion of autoreactive T cells in the central nervous systemInternational Immunology, 1995
- Effect of anti-interferon-γ monoclonal antibody treatment on the development of experimental allergic encephalomyelitis in resistant mouse strainsPublished by Elsevier ,1994
- Monoclonal Anti-Gamma Interferon Antibodies Enhance Experimental Allergic EncephalomyelitisAutoimmunity, 1993
- Apoptosis of αβ T lymphocytes in the nervous system in experimental autoimmune encephalomyelitis: Its possible implications for recovery and acquired toleranceJournal of Autoimmunity, 1992