Selective Pituitary Resistance to Thyroid Hormone Produced by Expression of a Mutant Thyroid Hormone Receptor β Gene in the Pituitary Gland of Transgenic Mice
- 7 April 1998
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 245 (1) , 204-210
- https://doi.org/10.1006/bbrc.1998.8396
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Thyroid Hormone Resistance Syndrome Manifests as an Aberrant Interaction between Mutant T3 Receptors and Transcriptional CorepressorsMolecular Endocrinology, 1997
- Mutations of CpG dinucleotides located in the triiodothyronine (T3)-binding domain of the thyroid hormone receptor (TR) beta gene that appears to be devoid of natural mutations may not be detected because they are unlikely to produce the clinical phenotype of resistance to thyroid hormone.Journal of Clinical Investigation, 1994
- Genetic analysis of 29 kindreds with generalized and pituitary resistance to thyroid hormone. Identification of thirteen novel mutations in the thyroid hormone receptor beta gene.Journal of Clinical Investigation, 1994
- The Variable Clinical Phenotype in Thyroid Hormone Resistance SyndromeThyroid®, 1994
- Identification of a novel mutation in the gene encoding the β‐triiodothyronine receptor in a patient with apparent selective pituitary resistance to thyroid hormoneClinical Endocrinology, 1993
- TEF, a transcription factor expressed specifically in the anterior pituitary during embryogenesis, defines a new class of leucine zipper proteins.Genes & Development, 1991
- Protein Factors in Thyrotropic Tumor Nuclear Extracts Bind to a Region of the Mouse Thyrotropin β-Subunit Promoter Essential for Expression in ThyrotropesMolecular Endocrinology, 1990
- TSH subunit gene promoters from a murine α-subunit producing tumor function normallyMolecular and Cellular Endocrinology, 1990
- Generalized resistance to thyroid hormone associated with a mutation in the ligand-binding domain of the human thyroid hormone receptor beta.Proceedings of the National Academy of Sciences, 1989
- Inappropriate Secretion of Thyroid-Stimulating HormoneAnnals of Internal Medicine, 1981