Apolipoprotein E deficiency promotes increased oxidative stress and compensatory increases in antioxidants in brain tissue
- 2 October 2002
- journal article
- research article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 33 (8) , 1115-1120
- https://doi.org/10.1016/s0891-5849(02)01001-8
Abstract
No abstract availableKeywords
This publication has 59 references indexed in Scilit:
- Amyloid-β: an antioxidant that becomes a pro-oxidant and critically contributes to Alzheimer’s diseaseFree Radical Biology & Medicine, 2001
- Homocysteine and lipid peroxidation in haemodialysis: role of folinic acid and vitamin ENephrology Dialysis Transplantation, 2001
- Homocysteine potentiates β‐amyloid neurotoxicity: role of oxidative stressJournal of Neurochemistry, 2001
- Could diet be used to reduce the risk of developing Alzheimer's disease?Medical Hypotheses, 1998
- Vitamins C, E and A and Heme Oxygenase in Rats Fed Methyl/Folate-Deficient Diets11This work was supported by NIH grant CA42710-10 and Westreco Inc., Van Nuys, CA.Free Radical Biology & Medicine, 1997
- Effects of α‐tocopherol and β‐carotene on hepatic lipid peroxidation and blood lipids in rats with dietary iron overloadNutrition and Cancer, 1996
- Increased Plasma and Lipoprotein Lipid Peroxidation in apo E-Deficient MiceBiochemical and Biophysical Research Communications, 1994
- Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGFBrain Research, 1993
- Generation of mice carrying a mutant apolipoprotein E gene inactivated by gene targeting in embryonic stem cells.Proceedings of the National Academy of Sciences, 1992
- Astrocytes synthesize apolipoprotein E and metabolize apolipoprotein E-containing lipoproteinsBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1987