Sensitive and specific assays for C3 nephritic factors clarify mechanisms underlying complement dysregulation
Open Access
- 1 November 2012
- journal article
- research article
- Published by Elsevier in Kidney International
- Vol. 82 (10) , 1084-1092
- https://doi.org/10.1038/ki.2012.250
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Causes of Alternative Pathway Dysregulation in Dense Deposit DiseaseClinical Journal of the American Society of Nephrology, 2012
- Common polymorphisms in C3, factor B, and factor H collaborate to determine systemic complement activity and disease riskProceedings of the National Academy of Sciences, 2011
- The disease-protective complement factor H allotypic variant Ile62 shows increased binding affinity for C3b and enhanced cofactor activityHuman Molecular Genetics, 2009
- Harmful molecular mechanisms in sepsisNature Reviews Immunology, 2008
- Translational Mini-Review Series on Complement Factor H: Renal diseases associated with complement factor H: novel insights from humans and animalsClinical and Experimental Immunology, 2008
- Molecular Dissection of Interactions between Components of the Alternative Pathway of Complement and Decay Accelerating Factor (CD55)Journal of Biological Chemistry, 2005
- Clinical Features and Metabolic and Autoimmune Derangements in Acquired Partial LipodystrophyMedicine, 2004
- Coupling complement regulators to immunoglobulin domains generates effective anti-complement reagents with extended half-lifein vivoClinical and Experimental Immunology, 2002
- On the origin of C3 nephritic factor (antibody to the alternative pathway C3 convertase): Evidence for the Adam and Eve concept of autoantibody productionClinical Immunology and Immunopathology, 1992
- Two types of C3 nephritic factor: Properdin-dependent C3NeF and properdin-independent C3NeFClinical Immunology and Immunopathology, 1990