Chest Pain in Heart-Transplant Recipients

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Abstract
THE article by Stark et al.1 in this issue of the Journal is an important addition to the evolving understanding of the physiology of the transplanted heart. These investigators have previously used measurements of the myocardial release of norepinephrine in response to the administration of tyramine to demonstrate the lack of such release in patients soon after cardiac transplantation, as well as the resumption of more normal release, believed to indicate at least partial sympathetic reinnervation, in later years in a high percentage of recipients.2 Although in the majority of animal models of heart transplantation early functional sympathetic reinnervation has . . .