NEURO-ARTHROPATHIES: A CONSIDERATION OF THE ETIOLOGY AND GENERAL CHARACTERISTICS

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Abstract
The association of bony changes—limited principally to joint structures—with grave disease of the central nervous system has been observed for a great number of years. The recognition of such structural osseous anomalies occurring in the course of tabes and syringomyelia was given impetus by Charcot, who was first to describe them accurately. Thus, the term Charcot joint found universal acceptance in the literature and was readily interpreted as a destructive arthritic process, dependent on and secondary to involvement in a disease process of certain fiber tracts in the spinal cord parenchyma. As tabes and syringomyelia were apparently the only affections that developed such arthropathies fairly consistently, it was readily assumed that they were peculiar to these diseases only and that such profound joint changes could develop on no other basis. The advent of the roentgen ray served further to emphasize the existence of these arthropathies, since the diagnosis could be

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