The DNA crosslink-induced S-phase checkpoint depends on ATR–CHK1 and ATR–NBS1–FANCD2 pathways
- 26 February 2004
- journal article
- Published by Springer Nature in The EMBO Journal
- Vol. 23 (5) , 1178-1187
- https://doi.org/10.1038/sj.emboj.7600113
Abstract
EMBO Press is an editorially independent publishing platform for the development of EMBO scientific publications.Keywords
This publication has 54 references indexed in Scilit:
- A novel ubiquitin ligase is deficient in Fanconi anemiaNature Genetics, 2003
- Sensing DNA Damage Through ATRIP Recognition of RPA-ssDNA ComplexesScience, 2003
- A splicing mutation affecting expression of ataxia–telangiectasia and Rad3–related protein (ATR) results in Seckel syndromeNature Genetics, 2003
- ATM phosphorylates p95/nbs1 in an S-phase checkpoint pathwayNature, 2000
- The Fanconi anaemia gene FANCF encodes a novel protein with homology to ROMNature Genetics, 2000
- Loss of the Fanconi anemia group C protein activity results in an inability to activate caspase-3 after ionizing radiationBiochimie, 2000
- ATAXIA-TELANGIECTASIA AND THE NIJMEGEN BREAKAGE SYNDROME: Related Disorders But Genes ApartAnnual Review of Genetics, 1997
- Correction: Cloning of cDNAs for Fanconi's anaemia by functional complementationNature, 1992
- Abnormal regulation of DNA replication and increased lethality in ataxia telangiectasia cells exposed to carcinogenic agents.1982
- The cell cycle of lymphocytes in Fanconi anemiaHuman Genetics, 1982