Phosphatidylcholine could be the source of 1,2-DAG which activates protein kinase C in EGF-stimulated colon carcinoma cells (HT29)
- 30 November 1995
- journal article
- Published by Elsevier in Cellular Signalling
- Vol. 7 (8) , 793-801
- https://doi.org/10.1016/0898-6568(95)02007-1
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Phosphatidylcholine breakdown and signal transductionBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1994
- Epidermal growth factor increases 32P incorporation into phosphatidylcholine and protein kinase C activity in colon carcinoma cell line (HT29)Cellular Signalling, 1993
- Control of Rat Granulosa Cell Mitosis by Phorbol Ester-, Cyclic AMP-, and Estradiol-17β-Dependent PathwaysBiology of Reproduction, 1993
- Effects of Epidermal Growth Factor versus Phorbol Ester on Kidney Epithelial (LLC-PK1) Tight Junction Permeability and Cell DivisionExperimental Cell Research, 1993
- Gene Modulation Accompanying Differentiation of Normal versus Malignant KeratinocytesExperimental Cell Research, 1993
- Evidence for the participation of protein kinase C and 3′,5′-cyclic AMP-dependent protein kinase in the stimulation of muscle cell proliferation by 1,25-dihydroxy-vitamin D3Molecular and Cellular Endocrinology, 1993
- Expression of TGF‐α/EGF and TGF‐p receptors in human colon carcinoma cell linesInternational Journal of Cancer, 1989
- Comparison of the Tyrosine Kinase Activity with the Proliferation Rate in Human Colon Solid Tumors and Tumor Cell LinesTumor Biology, 1988
- Polyphosphoinositide metabolism in adrenal glomerulosa cellsMolecular and Cellular Endocrinology, 1985
- Protein kinase C phosphorylation of the EGF receptor at a threonine residue close to the cytoplasmic face of the plasma membraneNature, 1984