Ligand-independent Activation of Fibroblast Growth Factor Receptors by Point Mutations in the Extracellular, Transmembrane, and Kinase Domains

This publication has 45 references indexed in Scilit:

• Constitutive Activation of Fibroblast Growth Factor Receptor-2 by a Point Mutation Associated with Crouzon Syndrome
  Published by Elsevier ,1995
• A recurrent mutation in the tyrosine kinase domain of fibroblast growth factor receptor 3 causes hypochondroplasia
  Nature Genetics, 1995
• Functions of fibroblast growth factors and their receptors
  Current Biology, 1995
• Alternately Spliced NH2-terminal Immunoglobulin-like Loop I in the Ectodomain of the Fibroblast Growth Factor (FGF) Receptor 1 Lowers Affinity for both Heparin and FGF-1
  Journal of Biological Chemistry, 1995
• Thanatophoric dysplasia (types I and II) caused by distinct mutations in fibroblast growth factor receptor 3
  Nature Genetics, 1995
• Apert syndrome results from localized mutations of FGFR2 and is allelic with Crouzon syndrome
  Nature Genetics, 1995
• Developmentally regulated expression of fibroblast growth factor receptor genes and splice variants by murine embryonic stem and embryonal carcinoma cells
  Developmental Genetics, 1994
• Production of Recombinant Xenopus Fibroblast Growth Factor Receptor-1 Using a Baculovirus Expression System
  Biochemical and Biophysical Research Communications, 1993
• Neu receptor dimerization
  Nature, 1989
• Multiple independent activations of the neu oncogene by a point mutation altering the transmembrane domain of p185
  Cell, 1986