A Novel IκB Protein, IκB-ζ, Induced by Proinflammatory Stimuli, Negatively Regulates Nuclear Factor-κB in the Nuclei
Open Access
- 1 July 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (29) , 27657-27662
- https://doi.org/10.1074/jbc.m103426200
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- MAIL, a novel nuclear IκB protein that potentiates LPS‐induced IL‐6 productionFEBS Letters, 2000
- TAK1 mediates an activation signal from toll‐like receptor(s) to nuclear factor‐κB in lipopolysaccharide‐stimulated macrophagesFEBS Letters, 2000
- Control of apoptosis by Rel/NF-κB transcription factorsOncogene, 1999
- Bcl3, an IκB Protein, Stimulates Activating Protein-1 Transactivation and Cellular ProliferationJournal of Biological Chemistry, 1999
- Bcl3, an IκB Protein, as a Novel Transcription Coactivator of the Retinoid X ReceptorPublished by Elsevier ,1998
- NF-κB AND REL PROTEINS: Evolutionarily Conserved Mediators of Immune ResponsesAnnual Review of Immunology, 1998
- MAP3K-related kinase involved in NF-KB induction by TNF, CD95 and IL-1Nature, 1997
- An Essential Role for NF-κB in Preventing TNF-α-Induced Cell DeathScience, 1996
- The p65 subunit of NF-kappa B regulates I kappa B by two distinct mechanisms.Genes & Development, 1993
- The oncoprotein Bcl-3 directly transactivates through κB motifs via association with DNA-binding p50B homodimersCell, 1993