The role of endogenous nitric oxide and platelet-activating factor in hypoxia-induced intestinal injury in rats
- 1 February 1994
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 106 (2) , 346-352
- https://doi.org/10.1016/0016-5085(94)90591-6
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Hypoxia increases stimulus-induced PAF production and release from human umbilical vein endothelial cellsBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1992
- Modulation of acute inflammation by endogenous nitric oxideEuropean Journal of Pharmacology, 1992
- Evidence nitric oxide mediates the vasodepressor response to hypoxia in sino-denervated ratsLife Sciences, 1992
- Effect of nitric oxide synthase inhibitors on hypotension in patients with septic shockThe Lancet, 1991
- Role of platelet activating factor and tumor necrosis factor-alpha in neonatal necrotizing enterocolitisThe Journal of Pediatrics, 1990
- Biological actions and properties of endothelium-derived nitric oxide formed and released from artery and vein.Circulation Research, 1989
- Role of oxygen derived free radicals in platelet activating factor induced bowel necrosis.Gut, 1988
- Necrotizing EnterocolitisNew England Journal of Medicine, 1984
- Platelet activating factor (PAF-acether) is released into rat pulmonary alveolar fluid as a consequence of hypoxiaBiochemical and Biophysical Research Communications, 1984
- The pathogenesis of ischemic gastroenterocolitis of the neonate: Selective gut mucosal ischemia in asphyxiated neonatal pigletsJournal of Pediatric Surgery, 1972