Antiinflammatory Effects of Soluble Complement Receptor Type 1 Promote Rapid Recovery of Ischemia/Reperfusion Injury in Rat Small Intestine
- 1 February 1999
- journal article
- Published by Elsevier in Clinical Immunology
- Vol. 90 (2) , 266-275
- https://doi.org/10.1006/clim.1998.4635
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Exacerbation of inflammation-associated colonic injury in rat through inhibition of cyclooxygenase-2.Journal of Clinical Investigation, 1996
- Induction of heat-shock protein 72 protects against ischemia/reperfusion in rat small intestineGastroenterology, 1995
- REGULATION OF THE COMPLEMENT CASCADE BY SOLUBLE COMPLEMENT RECEPTOR TYPE 1Transplantation, 1995
- Reperfusion injury after intestinal ischemiaCritical Care Medicine, 1993
- Blockade of Complement Activation Prevents Local and Pulmonary Albumin Leak After Lower Torso Ischemia—ReperfusionAnnals of Surgery, 1992
- Endothelial cell interactions with granulocytes: tethering and signaling moleculesImmunology Today, 1992
- THE EFFECT OF SOLUBLE COMPLEMENT RECEPTOR TYPE 1 ON HYPERACUTE XENOGRAFT REJECTIONTransplantation, 1991
- Soluble Human Complement Receptor Type 1: In Vivo Inhibitor of Complement Suppressing Post-Ischemic Myocardial Inflammation and NecrosisScience, 1990
- Neutrophil-mediated injury to endothelial cells. Enhancement by endotoxin and essential role of neutrophil elastase.Journal of Clinical Investigation, 1986
- Specific binding of leukotriene B4 to a receptor on human polymorphonuclear leukocytes.The Journal of Experimental Medicine, 1983