p130, p107, and pRb Are Differentially Regulated in Proliferating Cells and during Cell Cycle Arrest by α-Interferon
Open Access
- 1 September 1998
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 273 (37) , 23659-23667
- https://doi.org/10.1074/jbc.273.37.23659
Abstract
No abstract availableKeywords
This publication has 65 references indexed in Scilit:
- Modulation of E2F Activity Is Linked to Interferon-induced Growth Suppression of Hematopoietic CellsPublished by Elsevier ,1997
- A Role for the Cyclin-Dependent Kinase Inhibitor p21 in the G1 Cell Cycle Arrest Mediated by the Type I InterferonsJournal of Interferon & Cytokine Research, 1997
- Double-stranded RNA-dependent Protein Kinase Mediates c-Myc Suppression Induced by Type I InterferonsPublished by Elsevier ,1996
- Another role rolls inNature, 1996
- Interferon modulates the messenger RNA of G1-controlling genes to suppress the G1-to-S transition in Daudi cellsMolecular and Cellular Biochemistry, 1995
- Regulation of the retinoblastoma protein-related p107 by G1 cyclin complexes.Genes & Development, 1995
- Functional interaction between E2F-4 and p130: evidence for distinct mechanisms underlying growth suppression by different retinoblastoma protein family members.Genes & Development, 1995
- Interferon-α Inhibits Cyclin E- and Cyclin D1-Dependent CDK-2 Kinase Activity Associated with RB Protein and E2F in Daudi CellsBiochemical and Biophysical Research Communications, 1994
- Interferon treatment enhances the expression of underphosphorylated (biologically-active) retinoblastoma protein in human papilloma virus-infected cells through the inhibitory TGFβ1/IFNβ cytokine pathwayAntiviral Research, 1994
- Interaction of p107 with Cyclin A Independent of Complex Formation with Viral OncoproteinsScience, 1992