SPI-C and STAT6 can cooperate to stimulate IgE germline transcription
- 1 June 2006
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 344 (4) , 1155-1160
- https://doi.org/10.1016/j.bbrc.2006.04.026
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- Pub, a novel PU.1 binding protein, regulates the transcriptional activity of PU.1Biochemical and Biophysical Research Communications, 2003
- PU.1 and pRB Interact and Cooperate To Repress GATA-1 and Block Erythroid DifferentiationMolecular and Cellular Biology, 2003
- PU.1 is required for transcriptional activation of the Stat6 response element in the Igε promoterEuropean Journal of Immunology, 2003
- SPI-C, a PU-box binding ETS protein expressed temporarily during B-cell development and in macrophages, contains an acidic transactivation domain located to the N-terminusMolecular Immunology, 2003
- Genomic structure of mouse SPI-C and genomic structure and expression pattern of human SPI-CGene, 2002
- Physical and functional interactions between the transcription factor PU.1 and the coactivator CBPOncogene, 1999
- OCT proteins are qualitative rather than quantitative regulators of κ transcriptionMolecular Immunology, 1997
- Comparison of the Transactivation Domains of Stat5 and Stat6 in Lymphoid Cells and Mammary Epithelial CellsMolecular and Cellular Biology, 1997
- Activation of Transcription by PU.1 Requires both Acidic and Glutamine DomainsMolecular and Cellular Biology, 1996
- Effect of PU.1 phosphorylation on interaction with NF-EM5 and transcriptional activationScience, 1993