Involvement of c-Jun NH2-Terminal Kinase Pathway in Differential Regulation of Heat Shock Proteins by Anticancer Drugs
- 1 August 1999
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 262 (2) , 516-522
- https://doi.org/10.1006/bbrc.1999.1229
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Heat Shock Proteins Increase Resistance to ApoptosisExperimental Cell Research, 1996
- Pharmacological modulation of heat shock factor 1 by antiinflammatory drugs results in protection against stress-induced cellular damage.Proceedings of the National Academy of Sciences, 1995
- In vitro activation of purified human heat shock factor by heatBiochemistry, 1995
- Role of the Major Heat Shock Proteins as Molecular ChaperonesAnnual Review of Cell Biology, 1993
- Biological and Clinical Implications of Heat Shock Protein 27000 (Hsp27): a ReviewJNCI Journal of the National Cancer Institute, 1993
- Heat Shock Protein hsp70 in Patients With Axillary Lymph Node-Negative Breast Cancer: Prognostic ImplicationsJNCI Journal of the National Cancer Institute, 1993
- Activation of human heat shock genes is accompanied by oligomerization, modification, and rapid translocation of heat shock transcription factor HSF1.Molecular and Cellular Biology, 1993
- Cells in Stress: Transcriptional Activation of Heat Shock GenesScience, 1993
- Activation of heat shock gene transcription by heat shock factor 1 involves oligomerization, acquisition of DNA-binding activity, and nuclear localization and can occur in the absence of stress.Molecular and Cellular Biology, 1993
- Effect of Sodium Salicylate on the Human Heat Shock ResponseScience, 1992