A Polymorphism of the Human Matrix γ-Carboxyglutamic Acid Protein Promoter Alters Binding of an Activating Protein-1 Complex and Is Associated with Altered Transcription and Serum Levels
Open Access
- 1 August 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (35) , 32466-32473
- https://doi.org/10.1074/jbc.m104909200
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- AP-1 function and regulationPublished by Elsevier ,2002
- Vascular and valvar calcification: recent advancesHeart, 2001
- Matrix Gla Protein Is Regulated by a Mechanism Functionally Related to the Calcium-Sensing ReceptorBiochemical and Biophysical Research Communications, 2000
- Assay for Human Matrix Gla Protein in SerumArteriosclerosis, Thrombosis, and Vascular Biology, 2000
- Opposing activities of c-Fos and Fra-2 on AP-1 regulated transcriptional activity in mouse keratinocytes induced to differentiate by calcium and phorbol estersOncogene, 1997
- Spontaneous calcification of arteries and cartilage in mice lacking matrix GLA proteinNature, 1997
- High expression of genes for calcification-regulating proteins in human atherosclerotic plaques.Journal of Clinical Investigation, 1994
- jun-B inhibits and c-fos stimulates the transforming and trans-activating activities of c-junCell, 1989
- Jun-B differs in its biological properties from, and is a negative regulator of, c-JunCell, 1989
- Purified transcription factor AP-1 interacts with TPA-inducible enhancer elementsCell, 1987