EML4-ALK Rearrangement in Non-Small Cell Lung Cancer and Non-Tumor Lung Tissues
- 1 February 2009
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 174 (2) , 661-670
- https://doi.org/10.2353/ajpath.2009.080755
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- A mouse model for EML4-ALK -positive lung cancerProceedings of the National Academy of Sciences, 2008
- EML4-ALKFusion Gene and Efficacy of an ALK Kinase Inhibitor in Lung CancerClinical Cancer Research, 2008
- EML4–ALK fusion transcript is not found in gastrointestinal and breast cancersBritish Journal of Cancer, 2008
- Pathobiology of ALK+ anaplastic large-cell lymphomaBlood, 2007
- Identification of the transforming EML4–ALK fusion gene in non-small-cell lung cancerNature, 2007
- Identification of NVP-TAE684, a potent, selective, and efficacious inhibitor of NPM-ALKProceedings of the National Academy of Sciences, 2007
- Genomic Approaches to Lung CancerClinical Cancer Research, 2006
- Human EML4, a novel member of the EMAP family, is essential for microtubule formationExperimental Cell Research, 2006
- Activating Mutations in the Epidermal Growth Factor Receptor Underlying Responsiveness of Non–Small-Cell Lung Cancer to GefitinibNew England Journal of Medicine, 2004
- Fusion of a Kinase Gene, ALK , to a Nucleolar Protein Gene, NPM , in Non-Hodgkin's LymphomaScience, 1994