Discovery of a novel class of benzazepinone Nav1.7 blockers: Potential treatments for neuropathic pain
- 15 August 2007
- journal article
- research article
- Published by Elsevier in Bioorganic & Medicinal Chemistry Letters
- Vol. 17 (16) , 4630-4634
- https://doi.org/10.1016/j.bmcl.2007.05.076
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Loss‐of‐function mutations in the Nav1.7 gene underlie congenital indifference to pain in multiple human populationsClinical Genetics, 2007
- An SCN9A channelopathy causes congenital inability to experience painNature, 2006
- SCN9A Mutations in Paroxysmal Extreme Pain Disorder: Allelic Variants Underlie Distinct Channel Defects and PhenotypesNeuron, 2006
- The Role of Sodium Channels in Chronic Inflammatory and Neuropathic PainThe Journal of Pain, 2006
- Epidemiology of Refractory Neuropathic PainPain Practice, 2006
- Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neuronsBrain, 2005
- Functional Assay of Voltage-Gated Sodium Channels Using Membrane Potential-Sensitive DyesASSAY and Drug Development Technologies, 2004
- The Response of Neuropathic Pain and Pain in Complex Regional Pain Syndrome I to Carbamazepine and Sustained-Release Morphine in Patients Pretreated with Spinal Cord Stimulation: A Double-Blinded Randomized StudyAnesthesia & Analgesia, 2001
- The effect of intravenous lidocaine, tocainide, and mexiletine on spontaneously active fibers originating in rat sciatic neuromasPain, 1989
- Calcium Channel Receptor Binding Studies for Diltiazem and Its Major MetabolitesJournal of Cardiovascular Pharmacology, 1987