The γ-aminobutyric acid receptor γ3 subunit gene (GABRG3) is tightly linked to the α5 subunit gene (GABRA5) on human chromosome 15q11–q13 and is transcribed in the same orientation

This publication has 32 references indexed in Scilit:

• PARENTAL ORIGIN OF CHROMOSOME 15 DELETION IN PRADER-WILLI SYNDROME
  The Lancet, 2003
• Angelman syndrome associated with a maternal 15q11–13 deletion of less than 200 kb
  Human Molecular Genetics, 1994
• Phenotypic consequences of deletion of the gamma 3, alpha 5, or beta 3 subunit of the type A gamma-aminobutyric acid receptor in mice.
  Proceedings of the National Academy of Sciences, 1994
• Angelman syndrome.
  Journal of Medical Genetics, 1992
• Identification of a putative γ-aminobutyric acid (GABA) receptor subunit rho2 cDNA and colocalization of the genes encoding rho2 (GABRR2) and rho1 (GABRR1) to human chromosome 6q14–q21 and mouse chromosome 4
  Genomics, 1992
• The mouse pink-eyed dilution locus: a model for aspects of Prader-Willi syndrome, Angelman syndrome, and a form of hypomelanosis of Ito
  Mammalian Genome, 1992
• The syntenic relationship between the critical deletion region for the Prader-Willi/Angelman syndromes and proximal mouse chromosome 7
  Genomics, 1991
• Cloning of the gamma-aminobutyric acid (GABA) rho 1 cDNA: a GABA receptor subunit highly expressed in the retina.
  Proceedings of the National Academy of Sciences, 1991
• Microdissection of the Prader-Willi syndrome chromosome region and identification of potential gene sequences
  Genomics, 1990
• Chromosomal localization of GABAA receptor subunit genes: relationship to human genetic disease
  Neuron, 1989