p16INK4 is inactivated by extensive CpG methylation in human hepatocellular carcinoma

This publication has 15 references indexed in Scilit:

• Rapid quantitation of methylation differences at specific sites using methylation-sensitive single nucleotide primer extension (Ms-SNuPE)
  Nucleic Acids Research, 1997
• Frequency of Mutation and Deletion of the Tumor Suppressor Gene Cdkn2a (Mts1/P16) in Hepatocellular Carcinoma From An Australian Population
  Hepatology, 1997
• Methylation-specific PCR: a novel PCR assay for methylation status of CpG islands.
  Proceedings of the National Academy of Sciences, 1996
• Inactivation of P16Ink4 in Hepatocellular Carcinoma
  Hepatology, 1996
• Infrequent alterations of thep16INK4A gene in liver cancer
  International Journal of Cancer, 1996
• 5′ CpG island methylation is associated with transcriptional silencing of the tumour suppressor p16/CDKN2/MTS1 in human cancers
  Nature Medicine, 1995
• An efficient method for PCR analysis of mitochondrial DNA from paraffin-embedded archival heart tissue.
  Genome Research, 1995
• Dependence of transcriptional repression on CpG methylation density.
  Molecular and Cellular Biology, 1994
• A Cell Cycle Regulator Potentially Involved in Genesis of Many Tumor Types
  Science, 1994
• Deletions of the cyclin-dependent kinase-4 inhibitor gene in multiple human cancers
  Nature, 1994