Exposure of rat spinal neurones to NMDA, AMPA and kainate produces only short-term enhancements of responses to noxious and non-noxious stimuli
- 7 November 1994
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 181 (1-2) , 98-102
- https://doi.org/10.1016/0304-3940(94)90569-x
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Thyrotropin-releasing hormone (TRH)-induced facilitation of spinal neurotransmission: A role for NMDA receptorsNeuropharmacology, 1994
- Excitatory amino acid receptor‐mediated neurotransmission from cutaneous afferents in rat dorsal horn in vitro.The Journal of Physiology, 1993
- Acute mechanical hyperalgesia is produced by coactivation of AMPA and metabotropic glutamate receptorsNeuroReport, 1993
- Combined application of excitatory amino acids and substance P produces long-lasting changes in responses of primate spinothalamic tract neuronsBrain Research Reviews, 1993
- Central neural mediators of secondary hyperalgesia following heat injury in rats: Neuropeptides and excitatory amino acidsNeuroscience Letters, 1991
- Modification of the responses of primate spinothalamic neurons to mechanical stimulation by excitatory amino acids and an N-methyl-d-aspartate antagonistBrain Research, 1991
- Excitatory amino acid receptors, second messengers and regulation of intracellular Ca2+ in mammalian neuronsTrends in Pharmacological Sciences, 1990
- Excitatory amino acid receptors and nociceptive neurotransmission in rat spinal cordPain, 1990
- Mechanisms of blockade of excitatory amino acid receptor channelsTrends in Pharmacological Sciences, 1990
- The role of N-methylaspartate receptors in mediating responses of rat and cat spinal neurones to defined sensory stimuli.The Journal of Physiology, 1987