Genetic toxicology: chemical carcinogens modify DNA in a non-random fashion
- 31 December 1986
- journal article
- Published by Elsevier in Trends in Pharmacological Sciences
- Vol. 7, 354-357
- https://doi.org/10.1016/0165-6147(86)90385-8
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- Hypomethylation of ras oncogenes in primary human cancersPublished by Elsevier ,2005
- Expression of c-Ki-ras, c-Ha-ras, and c-myc in specific cell types during hepatocarcinogenesis.Molecular and Cellular Biology, 1985
- N-Sulfoöxy-2-aminofluorene is the major ultimate electrophilic and carcinogenic metabolite of N-hydroxy-2-acetylaminofluorene in the livers of infant male C57BL/6J × C3H/HeJ F1 (B6C3F1) miceCarcinogenesis: Integrative Cancer Research, 1985
- DNA-purine methylation in hepatic chromatin following exposure to dimethylnitrosamine or methylnitrosoureaBiochemical Pharmacology, 1984
- Non-random interaction of N-2-acetylaminofluorene and its N-hydroxy metabolite with DNA of rat hepatic parenchymal and non-parenchymal cell nuclei following in vivo administration of carcinogenChemico-Biological Interactions, 1984
- Induction of mammary carcinomas in rats by nitroso-methylurea involves malignant activation of H-ras-1 locus by single point mutationsNature, 1983
- Cell Specificity in DNA Binding and Repair of Chemical CarcinogensEnvironmental Health Perspectives, 1983
- Alkylation of DNA in specific hepatic chromatin fractions following exposure to methylnitrosourea or dimethylnitrosamineToxicology and Applied Pharmacology, 1981
- Non-random nature of 2-acetylaminofluorene-induced alterations of DNA template capacityChemico-Biological Interactions, 1979
- Quantitative and qualitative aspects of the binding of N-hydroxy-2-acetylaminofluorene to hepatic chromatin fractionsChemico-Biological Interactions, 1979