An increase in the ATP levels occurs in cerebellar granule cells en route to apoptosis in which ATP derives from both oxidative phosphorylation and anaerobic glycolysis
- 7 February 2005
- journal article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Bioenergetics
- Vol. 1708 (1) , 50-62
- https://doi.org/10.1016/j.bbabio.2005.01.009
Abstract
No abstract availableKeywords
This publication has 66 references indexed in Scilit:
- Role of N-terminal tau domain integrity on the survival of cerebellar granule neuronsCell Death & Differentiation, 2003
- Role of oxidative stress in the apoptotic cell death of cultured cerebellar granule neuronsJournal of Neuroscience Research, 2001
- Rapid uncoupling of oxidative phosphorylation accompanies glutamate toxicity in rat cerebellar granule cellsNeuroReport, 1996
- mtDNA‐depleted U937 cells are sensitive to TNF and Fas‐mediated cytototxicityFEBS Letters, 1995
- Neurotrophic effects of NMDA receptor activation on developing cerebellar granule cellsJournal of Neurocytology, 1993
- Bcl-2 blocks apoptosis in cells lacking mitochondrial DNANature, 1993
- Cardiac metabolism - emergence, decline, and resurgence. Part ICardiovascular Research, 1992
- Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell deathNature, 1990
- Lactate release from cultured astrocytes and neurons: A comparisonGlia, 1988
- Citrate inhibition of phosphofructokinase and the Pasteur effectBiochemical and Biophysical Research Communications, 1965