10q(q23?qter) duplication: GOTs, HK1, and other gene markers

Abstract
Gene marker analyses have been carried out in a patient with 10q(q23→qter) duplication. The observed elevation of red cell glutamic oxaloacetic transaminase activity is compatible with earlier somatic cell hybridization studies that mapped the locus to this region. Hexokinase-1 activity in the red cells was normal, which is consistent with its prior assignment to the unaffected part of chromosome 10 (10pter→q23).