Transforming Growth Factor β1 Induces Nuclear Export of Inhibitory Smad7
Open Access
- 1 November 1998
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 273 (44) , 29195-29201
- https://doi.org/10.1074/jbc.273.44.29195
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Physical and Functional Interaction of Murine and Xenopus Smad7 with Bone Morphogenetic Protein Receptors and Transforming Growth Factor-β ReceptorsJournal of Biological Chemistry, 1998
- Human Smad3 and Smad4 Are Sequence-Specific Transcription ActivatorsPublished by Elsevier ,1998
- Dual role of the Smad4/DPC4 tumor suppressor in TGFβ-inducible transcriptional complexesGenes & Development, 1997
- TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4The EMBO Journal, 1997
- TGF-β signalling through the Smad pathwayTrends in Cell Biology, 1997
- Partnership between DPC4 and SMAD proteins in TGF-β signalling pathwaysNature, 1996
- Receptor-associated Mad homologues synergize as effectors of the TGF-β responseNature, 1996
- MADR2 Maps to 18q21 and Encodes a TGFβ–Regulated MAD–Related Protein That Is Functionally Mutated in Colorectal CarcinomaCell, 1996
- A human Mad protein acting as a BMP-regulated transcriptional activatorNature, 1996
- The TGF-beta superfamily: new members, new receptors, and new genetic tests of function in different organisms.Genes & Development, 1994