AR-R15896AR, a low affinity, use-dependent, NMDA antagonist, is protective in a primate model of stroke
- 31 December 2000
- journal article
- abstracts
- Published by Elsevier in Journal Of Stroke & Cerebrovascular Diseases
- Vol. 9 (6) , 303-312
- https://doi.org/10.1053/jscd.2000.18738
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Clomethiazole protects against hemineglect in a primate model of strokeBrain Research Bulletin, 2000
- The Low‐Affinity, Use‐Dependent NMDA Receptor Antagonist AR‐R 15896AR: An Update of Progress in StrokeAnnals of the New York Academy of Sciences, 1999
- Efficacy of AR-R15896AR in the rat monofilament model of transient middle cerebral artery occlusionJournal Of Stroke & Cerebrovascular Diseases, 1999
- Functional Benefit from Clomethiazole Treatment after Focal Cerebral Ischemia in a Nonhuman Primate SpeciesExperimental Neurology, 1999
- Lippincott‐Raven Publishers, Philadelphia © International League Against Epilepsy Remacemide Hydrochloride and ARL 15896AR Lack Abuse Potential: Additional Differences from Other Uncompetitive NMDA AntagonistsEpilepsia, 1996
- Effects of anticonvulsants in a novel operant learning paradigm in rats: Comparison of remacemide hydrochloride and FPL 15896AR to other anticonvulsant agentsEpilepsy Research, 1995
- Focal Cerebral Ischaemia in the Cat: Treatment with the Glutamate Antagonist MK-801 after Induction of IschaemiaJournal of Cerebral Blood Flow & Metabolism, 1988
- Glutamate Antagonist Therapy Reduces Neurologic Deficits Produced by Focal Central Nervous System IschemiaArchives of Neurology, 1988
- Protective Effect of the Glutamate Antagonist, MK-801 in Focal Cerebral Ischemia in the CatJournal of Cerebral Blood Flow & Metabolism, 1988
- Systemic administration of MK-801 protects against ischemia-induced hippocampal neurodegeneration in the gerbilJournal of Neuroscience, 1987