Mac-1 Signaling via Src-Family and Syk Kinases Results in Elastase-Dependent Thrombohemorrhagic Vasculopathy
- 1 August 2006
- Vol. 25 (2) , 271-283
- https://doi.org/10.1016/j.immuni.2006.05.014
Abstract
No abstract availableKeywords
This publication has 62 references indexed in Scilit:
- SLP-76 Regulates Fcγ Receptor and Integrin Signaling in NeutrophilsImmunity, 2003
- Expression and tyrosine phosphorylation of Cbl regulates macrophage chemokinetic and chemotactic movementJournal of Cellular Physiology, 2003
- Regulation of the Adhesion versus Cytotoxic Functions of the Mac-1/CR3/αMβ2 - lntegrin GlycoproteinCritical Reviews in Immunology, 2000
- Gelatinase B–deficient Mice Are Resistant to Experimental Bullous PemphigoidThe Journal of Experimental Medicine, 1998
- A Role for Mac-1 (CDIIb/CD18) in Immune Complex–stimulated Neutrophil Function In Vivo: Mac-1 Deficiency Abrogates Sustained Fcγ Receptor–dependent Neutrophil Adhesion and Complement-dependent Proteinuria in Acute GlomerulonephritisThe Journal of Experimental Medicine, 1997
- TNF-α stimulates the biosynthesis of complement C3 and factor B by human umbilical cord vein endothelial cellsCancer Letters, 1997
- Deficiency of Src family kinases p59/61hck and p58c-fgr results in defective adhesion-dependent neutrophil functions.The Journal of cell biology, 1996
- Cell surface-bound elastase and cathepsin G on human neutrophils: a novel, non-oxidative mechanism by which neutrophils focus and preserve catalytic activity of serine proteinases.The Journal of cell biology, 1995
- Macrophages form circular zones of very close apposition to lgG‐Coated surfacesCell Motility, 1990
- Complement Activation during Cardiopulmonary BypassNew England Journal of Medicine, 1981