Plasma from myasthenia gravis patients reduces acetylcholine receptor agonist-induced Na+ flux into TE671 cell line
- 1 August 1988
- journal article
- research article
- Published by Elsevier in Journal of Neuroimmunology
- Vol. 19 (1-2) , 141-148
- https://doi.org/10.1016/0165-5728(88)90043-4
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Immunological and pharmacological heterogeneity of α-bungarotoxin binding sites extracted from TE671 cellsJournal of Neuroimmunology, 1988
- Using Monoclonal Antibodies to Determine the Structures of Acetylcholine Receptors from Electric Organs, Muscles, and NeuronsaAnnals of the New York Academy of Sciences, 1987
- Functional Inhibition of Acetylcholine Receptors by Antibodies in Myasthenic SeraAnnals of the New York Academy of Sciences, 1987
- Characterization of Curaremimetic Neurotoxin Binding Sites on Membrane Fractions Derived from the Human Medulloblastoma Clonal Line, TE671Journal of Neurochemistry, 1986
- Acetylcholine receptor antibody as a diagnostic test for myasthenia gravis: results in 153 validated cases and 2967 diagnostic assays.Journal of Neurology, Neurosurgery & Psychiatry, 1985
- Paraneoplastic myasthenic syndrome IgG inhibits 45Ca2+ flux in a human small cell carcinoma lineNature, 1985
- Effects of monoclonal antibodies on the function of acetylcholine receptors purified from Torpedo californica and reconstituted into vesiclesBiochemistry, 1985
- Myasthenia gravis and myasthenic syndromesAnnals of Neurology, 1984
- Functional Activities of Autoantibodies to Acetylcholine Receptors and the Clinical Severity of Myasthenia GravisNew England Journal of Medicine, 1982
- Establishment of a human medulloblastoma cell lineInternational Journal of Cancer, 1977