Diquat induces renal proximal tubule injury in glutathione reductase-deficient mice

This publication has 20 references indexed in Scilit:

• Role of glutathione transport processes in kidney function
  Toxicology and Applied Pharmacology, 2004
• Oxidative stress in end-stage renal disease: an emerging threat to patient outcome
  Nephrology Dialysis Transplantation, 2003
• Expression of somatostatin receptors 3, 4, and 5 in mouse kidney proximal tubules
  Kidney International, 2003
• Effects of Fasting on Tissue Contents of Coenzyme A and Related Intermediates in Rats
  Pediatric Research, 2002
• Cellular Glutathione Peroxidase Protects Mice Against Lethal Oxidative Stress Induced by Various Doses of Diquat
  Proceedings of the Society for Experimental Biology and Medicine, 1999
• Knockout of cellular glutathione peroxidase gene renders mice susceptible to diquat-induced oxidative stress
  Free Radical Biology & Medicine, 1999
• Selenium‐dependent cellular glutathione peroxidase protects mice against a pro‐oxidant‐induced oxidation of NADPH, NADH, lipids, and protein
  The FASEB Journal, 1999
• Increased repair of O⁶-alkylguanine DNA adducts in glioma-derived human cells resistant to the cytotoxic and cytogenetic effects of 1, 3-bis(2-chloroethyl)-1-nitrosourea
  Carcinogenesis: Integrative Cancer Research, 1986
• Biliary excretion of glutathione and glutathione disulfide in the rat. Regulation and response to oxidative stress.
  Journal of Clinical Investigation, 1984
• Liver Necrosis and Lipid Peroxidation in the Rat as the Result of Paraquat and Diquat Administration
  Journal of Clinical Investigation, 1980