Imprinting of the MEA Polycomb gene is controlled by antagonism between MET1 methyltransferase and DME glycosylase.
Open Access
- 31 December 2003
- journal article
- Published by Elsevier
- Vol. 5 (6) , 891-901
- https://doi.org/10.1016/s1534-5807(03)00361-7
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- ThePolycomb-group protein MEDEA regulates seed development by controlling expression of the MADS-box genePHERES1Genes & Development, 2003
- Chromatin modification and epigenetic reprogramming in mammalian developmentNature Reviews Genetics, 2002
- Interplay between Two Epigenetic MarksCurrent Biology, 2002
- An APE that proofreadsNature, 2002
- Imprinting and the Epigenetic Asymmetry Between Parental GenomesScience, 2001
- DNA Methylation and Epigenetic Inheritance in Plants and Filamentous FungiScience, 2001
- Genetic conflict, genomic imprinting and establishment of the epigenotype in relation to growthReproduction, 2001
- Mobilization of transposons by a mutation abolishing full DNA methylation in ArabidopsisNature, 2001
- Mutations in the FIE and MEA Genes That Encode Interacting Polycomb Proteins Cause Parent-of-Origin Effects on Seed Development by Distinct MechanismsPlant Cell, 2000
- Development of endosperm in Arabidopsis thalianaSexual Plant Reproduction, 1999