Activation of Maxi Cl− channels by antiestrogens and phenothiazines in NIH3T3 fibroblasts
- 31 May 2002
- Vol. 67 (6) , 439-445
- https://doi.org/10.1016/s0039-128x(01)00174-x
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Nongenomic actions of estrogens and xenoestrogens by binding at a plasma membrane receptor unrelated to estrogen receptor α and estrogen receptor βProceedings of the National Academy of Sciences, 2000
- Estrogen Modulates a Large Conductance Chloride Channel in Cultured Porcine Aortic Endothelial CellsJournal of Cardiovascular Pharmacology, 2000
- Non‐genomic actions of 17β‐oestradiol in mouse pancreatic β‐cells are mediated by a cGMP‐dependent protein kinaseThe Journal of Physiology, 1999
- Acute Activation of Maxi-K Channels ( hSlo ) by Estradiol Binding to the β SubunitScience, 1999
- Rapid insulinotropic effect of 17β‐estradiol via a plasma membrane receptorThe FASEB Journal, 1998
- Environmental estrogenic pollutants induce acute vascular relaxation by inhibiting L‐type Ca 2+ channels in smooth muscle cellsThe FASEB Journal, 1998
- Steroid hormone receptors: Many Actors in search of a plotCell, 1995
- Novel plasma membrane action of estrogen and antiestrogens revealed by their regulation of a large conductance chloride channelThe FASEB Journal, 1994
- Electrophysiological evidence for a rapid membrane action of the gonadal steroid, 17β-estradiol, on CA1 pyramidal neurons of the rat hippocampusBrain Research, 1991
- 17β-Estradiol depolarization of hypothalamic neurons is mediated by cyclic AMPBrain Research, 1990