The quantitation of altered hepatic foci during multistage hepatocarcinogenesis in the rat: Transforming growth factor α expression as a marker for the stage of progression

This publication has 35 references indexed in Scilit:

• Abundant TGFα a precursor and EGF receptor expression as a possible mechanism for the preferential growth of carcinogeninduced preneoplastic and neoplastic hepatocytes in rats
  Carcinogenesis: Integrative Cancer Research, 1994
• Effects of polychlorinated biphenyls in rat liver: Quantitative analysis of enzyme-altered foci
  Toxicology and Applied Pharmacology, 1991
• Effects of a Choline-deficient Diet and a Hypolipidemic Agent on Single GlutathioneS-Transferase Placental Form-positive Hepatocytes in Rat Liver
  Japanese Journal of Cancer Research, 1990
• Sequential Changes in Epidermal Growth Factor Receptor/Ligand Function in Cultured Rat Liver Epithelial Cells Transformed Chemically in vitro
  Pathobiology, 1990
• Progression: The Terminal Stage in Carcinogenesis
  Japanese Journal of Cancer Research, 1989
• Single GST-P positive liver cells — putative initiated. hepatocytes
  Carcinogenesis: Integrative Cancer Research, 1987
• Quantitative stereological evaluation of four histochemical markers of altered foci in multistage hepatocarcinogenesis in the rat
  Carcinogenesis: Integrative Cancer Research, 1987
• Malignant progression of mouse skin papillomas treated with ethylnitrosourea, N-methyl-N′-nitro-N-nitrosoguanidine, or 12-O-tetradecanoylphorbol-13-acetate
  Cancer Letters, 1986
• Karyotype evolution and tumor development
  Cancer Genetics and Cytogenetics, 1983
• Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures.
  Journal of Histochemistry & Cytochemistry, 1981