Ipaf is upregulated by tumor necrosis factor‐α in human leukemia cells

Abstract
Ipaf has been associated with apoptosis, cytokine processing, and nuclear factor-κB activation. Here, we describe that Ipaf is highly expressed in myelomonocytic cells and that the mRNA levels of Ipaf progressively increase during differentiation of CD34 + progenitors to granulocytes and monocytes. Additionally, treatment with tumor necrosis factor-α and exposure to UV radiation induced the transcriptional activation of Ipaf in human leukemia HL-60 cells. Thus, Ipaf may contribute to modulate the response of myeloid cells to genotoxic and pro-inflammatory stimuli.