Mechanism of action of glucocorticosteroid hormones: possible implications for therapy of neuroimmunological disorders

This publication has 50 references indexed in Scilit:

• Caspase structure, proteolytic substrates, and function during apoptotic cell death
  Cell Death & Differentiation, 1999
• Impairment of TNF-Receptor-1 Signaling but not Fas Signaling Diminishes T-Cell Apoptosis in Myelin Oligodendrocyte Glycoprotein Peptide-Induced Chronic Demyelinating Autoimmune Encephalomyelitis in Mice
  The American Journal of Pathology, 1999
• Corticosteroid treatment of experimental autoimmune encephalomyelitis in the Lewis rat results in loss of Vβ8.2+ and myelin basic protein-reactive cells from the spinal cord, with increased total T-cell apoptosis but reduced apoptosis of Vβ8.2+ cells
  Journal of Neuroimmunology, 1996
• Apoptosis of T cells and Macrophages in the Central Nervous System of Intact and Adrenalectomized Lewis Rats During Experimental Allergic Encephalomyelitis
  Journal of Autoimmunity, 1996
• Antigen presentation by astrocytes primes rat T lymphocytes for apoptotic cell death
  Brain, 1996
• Secretion of Nitrite by Schwann Cells and Its Effect on T-Cell Activationin Vitro
  Cellular Immunology, 1996
• Human astrocytes are only partially competent antigen presenting cells
  Brain, 1994
• Phagocyte recognition of cells undergoing apoptosis
  Immunology Today, 1993
• A Randomized, Controlled Trial of Methylprednisolone or Naloxone in the Treatment of Acute Spinal-Cord Injury
  New England Journal of Medicine, 1990
• Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation
  Nature, 1980