Effects of N-acetyl-l-cysteine on T-cell apoptosis are not mediated by increased cellular glutathione
- 10 March 1995
- journal article
- Published by Elsevier in Immunology Letters
- Vol. 45 (3) , 205-209
- https://doi.org/10.1016/0165-2478(95)00004-o
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Oxidative stress as a mediator of apoptosisPublished by Elsevier ,2002
- Protease inhibitors selectively block T cell receptor-triggered programmed cell death in a murine T cell hybridoma and activated peripheral T cells.The Journal of Experimental Medicine, 1993
- Bcl-2 functions in an antioxidant pathway to prevent apoptosisCell, 1993
- N‐Acetylcysteine inhibits apoptosis and decreases viral particles in HIV‐chronically infected U937 cellsFEBS Letters, 1993
- Apoptosis and diseaseThe Lancet, 1993
- Rescue of thymocytes and T cell hybridomas from glucocorticoid‐induced apoptosis by stimulation via the T cell receptor/CD3 complex: a possible in vitro model for positive selection of the T cell repertoireEuropean Journal of Immunology, 1991
- Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell deathNature, 1990
- Redox Regulation of Fos and Jun DNA-Binding Activity in VitroScience, 1990
- 2,3,7,8-Tetrachlorodibenzo- p -Dioxin Kills Immature Thymocytes by Ca 2+ -Mediated Endonuclease ActivationScience, 1988
- High-performance liquid chromatography analysis of nanomole levels of glutathione, glutathione disulfide, and related thiols and disulfidesAnalytical Biochemistry, 1980