Chronic ethanol treatment upregulates the NMDA receptor function and binding in mammalian cortical neurons
- 30 June 1995
- journal article
- Published by Elsevier in Molecular Brain Research
- Vol. 30 (2) , 347-356
- https://doi.org/10.1016/0169-328x(95)00019-o
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Enhancement of NMDA toxicity and calcium responses by chronic exposure of cultured cortical neurons to ethanolNeuroscience Letters, 1994
- Chronic Ethanol Exposure Potentiates NMDA Excitotoxicity in Cerebral Cortical NeuronsJournal of Neurochemistry, 1993
- Ethanol Inhibits NMDA Receptor‐Mediated Excitotoxicity in Rat Primary Neuronal CulturesAlcohol, Clinical and Experimental Research, 1993
- Drug discrimination analysis of ethanol as an N-methyl-D-aspartate receptor antagonistEuropean Journal of Pharmacology, 1992
- NMDA Receptor Complex Antagonists Have Ethanol‐like Discriminative Stimulus EffectsAnnals of the New York Academy of Sciences, 1992
- Acute and Chronic Ethanol Exposure Alters the Function of Hippocampal Kainate Receptors Expressed in Xenopus OocytesJournal of Neurochemistry, 1992
- Mechanism of Inhibition of N‐Methyl‐d‐Aspartate‐Stimulated Increases in Free Intracellular Ca2+ Concentration by EthanolJournal of Neurochemistry, 1991
- The effects of chronic ethanol exposure on N-methyl-d-aspartate-stimulated overflow of [3H]catecholamines from rat brainBrain Research, 1991
- Ethanol inhibits NMDA-induced increases in free intracellular Ca2+ in dissociated brain cellsBrain Research, 1989
- Increased dihydropyridine-sensitive calcium channels in rat brain may underlie ethanol physical dependenceNeuropharmacology, 1987