Overexpression of cdk Inhibitor p16INK4aby Adenovirus Vector Inhibits Cardiac Hypertrophy in vitro and in vivo: a Novel Strategy for the Gene Therapy of Cardiac Hypertrophy
- 31 August 2001
- journal article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 33 (8) , 1493-1504
- https://doi.org/10.1006/jmcc.2001.1412
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
- Ministry of Health, Labour and Welfare
This publication has 39 references indexed in Scilit:
- Cancer Cell CyclesScience, 1996
- Another role rolls inNature, 1996
- Endothelin-3 induces hypertrophy of cardiomyocytes by the endogenous endothelin-1-mediated mechanism.Journal of Clinical Investigation, 1996
- Natriuretic peptides inhibit angiotensin II-induced proliferation of rat cardiac fibroblasts by blocking endothelin-1 gene expression.Journal of Clinical Investigation, 1995
- Tumour-derived p16 alleles encoding proteins defective in cell-cycle inhibitionNature, 1995
- Inhibitors of mammalian G1 cyclin-dependent kinases.Genes & Development, 1995
- The retinoblastoma protein and cell cycle controlCell, 1995
- Activity of RNA polymerase I transcription factor UBF blocked by Rb gene productNature, 1995
- Autocrine release of angiotensin II mediates stretch-induced hypertrophy of cardiac myocytes in vitroCell, 1993
- S. cerevisiae TFIIIB is the transcription initiation factor proper of RNA polymerase III, while TFIIIA and TFIIIC are assembly factorsCell, 1990