Vascular Endothelial Growth Factor (VEGF)-A165-induced Prostacyclin Synthesis Requires the Activation of VEGF Receptor-1 and -2 Heterodimer
Open Access
- 1 March 2005
- journal article
- Published by Elsevier
- Vol. 280 (11) , 9904-9912
- https://doi.org/10.1074/jbc.m412017200
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Simvastatin potenciates PGI2 release induced by HDL in human VSMC: effect on Cox-2 up-regulation and MAPK signalling pathways activated by HDLAtherosclerosis, 2004
- HDL 3 Induces Cyclooxygenase-2 Expression and Prostacyclin Release in Human Endothelial Cells Via a p38 MAPK/CRE-Dependent Pathway: Effects on COX-2/PGI-Synthase CouplingArteriosclerosis, Thrombosis, and Vascular Biology, 2004
- Mechanisms involved in the stimulation of prostacyclin synthesis by human lymphocytes in human umbilical vein endothelial cellsBritish Journal of Pharmacology, 2003
- Role of cyclooxygenase 2, p38 and p42/44 MAPK in the secretion of prostacyclin induced by epidermal growth factor, endothelin-1 and angiotensin II in rat ventricular cardiomyocytesJournal of Molecular and Cellular Cardiology, 2003
- Relative effects of VEGF‐A and VEGF‐C on endothelial cell proliferation, migration and PAF synthesis: Role of neuropilin‐1Journal of Cellular Biochemistry, 2002
- Receptor Chimeras Indicate That the Vascular Endothelial Growth Factor Receptor-1 (VEGFR-1) Modulates Mitogenic Activity of VEGFR-2 in Endothelial CellsJournal of Biological Chemistry, 2000
- SB 203580 is a specific inhibitor of a MAP kinase homologue which is stimulated by cellular stresses and interleukin‐1Published by Wiley ,2000
- The Vascular Endothelial Growth Factor Receptor Flt-1 Mediates Biological ActivitiesJournal of Biological Chemistry, 1996
- Cytokine-activated endothelial cells express an isotype of nitric oxide synthase which is tetrahydrobiopterin-dependent, calmodulin-independent and inhibited by arginine analogs with a rank-order of potency characteristic of activated macrophagesBiochemical and Biophysical Research Communications, 1991
- Angiogenic FactorsScience, 1987