NIRF induces G1 arrest and associates with Cdk2
- 1 June 2004
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 319 (2) , 464-468
- https://doi.org/10.1016/j.bbrc.2004.04.190
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- NIRF, a novel RING finger protein, is involved in cell-cycle regulationBiochemical and Biophysical Research Communications, 2002
- Involvement of Myc Activity in a G1/S-Promoting Mechanism Parallel to the pRb/E2F PathwayMolecular and Cellular Biology, 2000
- Cloning and mapping of Np95 gene which encodes a novel nuclear protein associated with cell proliferationMammalian Genome, 1998
- Role of the Rb/E2F pathway in cell growth controlJournal of Cellular Physiology, 1997
- Repression of c-Myc responsive genes in cycling cells causes G1 arrest through reduction of cyclin E/CDK2 kinase activityOncogene, 1997
- The Retinoblastoma Protein: A Master Regulator of Cell Cycle, Differentiation and ApoptosisEuropean Journal of Biochemistry, 1997
- Cyclin E-induced S phase without activation of the pRb/E2F pathway.Genes & Development, 1997
- Inhibition of E2F Activity by the Cyclin-Dependent Protein Kinase Inhibitor p21 in Cells Expressing or Lacking a Functional Retinoblastoma ProteinMolecular and Cellular Biology, 1996
- Different Roles for Cyclins D1 and E in Regulation of the G1-to-S TransitionMolecular and Cellular Biology, 1995
- Human Cyclin E, a Nuclear Protein Essential for the G1-to-S Phase TransitionMolecular and Cellular Biology, 1995