Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
- 1 August 1993
- Vol. 74 (4) , 609-619
- https://doi.org/10.1016/0092-8674(93)90509-o
Abstract
No abstract availableThis publication has 48 references indexed in Scilit:
- bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell deathCell, 1993
- Cell death genes in invertebrates and (maybe) vertebratesCurrent Opinion in Neurobiology, 1993
- Mad: A heterodimeric partner for Max that antagonizes Myc transcriptional activityCell, 1993
- Mechanisms and Functions of Cell DeathAnnual Review of Cell Biology, 1991
- bcl-2 inhibits multiple forms of apoptosis but not negative selection in thymocytesCell, 1991
- Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell deathNature, 1990
- Basic local alignment search toolJournal of Molecular Biology, 1990
- A conformational preference parameter to predict helices in integral membrane proteinsBiochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology, 1986
- Cloning the chromosomal breakpoint of t(14;18) human lymphomas: clustering around Jh on chromosome 14 and near a transcriptional unit on 18Cell, 1985
- CELL DEATHS IN NORMAL VERTEBRATE ONTOGENYBiological Reviews, 1951