Analysis of the NF-κB and PI 3-Kinase/Akt Survival Pathways in Nerve Growth Factor-Dependent Neurons

This publication has 63 references indexed in Scilit:

• Glycogen Synthase Kinase-3β Activity Is Critical for Neuronal Death Caused by Inhibiting Phosphatidylinositol 3-Kinase or Akt but Not for Death Caused by Nerve Growth Factor Withdrawal
  Journal of Biological Chemistry, 2000
• The Rel/NF-κB Family Directly Activates Expression of the Apoptosis Inhibitor Bcl-x_L
  Molecular and Cellular Biology, 2000
• Involvement of regulatory and catalytic subunits of phosphoinositide 3-kinase in NF-κB activation
  Proceedings of the National Academy of Sciences, 1999
• Phosphatidylinositol 3-Kinase and Akt Protein Kinase Are Necessary and Sufficient for the Survival of Nerve Growth Factor-Dependent Sympathetic Neurons
  Journal of Neuroscience, 1998
• Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery
  Cell, 1997
• Suppression of tumor necrosis factor-induced cell death by inhibitor of apoptosis c-IAP2 is under NF-κB control
  Proceedings of the National Academy of Sciences, 1997
• In sympathetic but not sensory neurones, phosphoinositide-3 kinase is important for NGF-dependent survival and the retrograde transport of 125I-βNGF
  Brain Research, 1997
• Selective Activation of NF-κB by Nerve Growth Factor Through the Neurotrophin Receptor p75
  Science, 1996
• Coupling of a Signal Response Domain in IκBα to Multiple Pathways for NF-κB Activation
  Molecular and Cellular Biology, 1995
• Control of IκB-α Proteolysis by Site-Specific, Signal-Induced Phosphorylation
  Science, 1995